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THE CRAZY LIFE AND DEATH OF HOWARD HUGHES

Howard Hughes was a man who could design and test-fly an airplane, direct a movie, seduce a starlet, buy casino hotels, disappear for years, and still make headlines without showing his face. He was as much a symbol of American ambition as he was a cautionary tale of what unchecked wealth, genius, and madness can do to a man. Born into privilege, fueled by obsession, and haunted by demons, Hughes lived a life so extreme that it bordered on mythology. But his death—quiet, grim, and mysterious—might be stranger than the intense living that led to it. Here’s the drama of the crazy life and death of Howard Hughes.

To understand his end, we have to rewind to the beginning of a life lived on the edges of brilliance and breakdown. Howard Hughes was many things: inventor, aviator, filmmaker, billionaire, recluse, suspected intelligence asset, and perhaps most tragically, a prisoner of his own mind.

He died aboard a private jet, his six-foot-four frame weighing only ninety pounds, unrecognizable even to those who’d once worshipped him. The official version says kidney failure. But the deeper you dig, the more the story starts to crack. It was a death as strange as his life—one that still casts a long shadow.

Howard Robard Hughes Jr. was born on December 24, 1905, in Humble, Texas, into a family drenched in oil money. His father, Howard Sr., invented the Hughes rotary drill bit and founded the Hughes Tool Company, which would bankroll young Howard’s endless stream of curiosities and obsessions. By age 11, he built Houston’s first wireless radio transmitter. At 12, he constructed a motorized bicycle from scrap parts. By 14, he was designing working aircraft models in his room. But early brilliance often walks hand in hand with isolation.

Tragedy struck fast and deep. His mother Allene died when he was just 16—reportedly from complications of an ectopic pregnancy. His father died suddenly two years later from a heart attack. At 18, Hughes was a billionaire orphan with complete control over the Hughes Tool fortune. No advisors. No parental guidance. Just money, ambition, and a ticking mind that was already showing cracks.

He dropped out of Rice University and headed west to Los Angeles. Hollywood in the 1920s was wild, wide open, and vulnerable to someone like Hughes: rich, eccentric, and hungry to create. His first film, “Swell Hogan,” was a bomb. But he rebounded with Hell’s Angels, an over-the-top war epic that cost $4 million, used real WWI aircraft, and took three years to complete. Hughes delayed filming repeatedly, waiting for perfect cloud formations to shoot aerial scenes. That level of obsessive control would become his hallmark.

He followed up with The Outlaw (1943), mostly remembered for its promotional posters featuring Jane Russell’s cleavage. Hughes engineered a custom bra for her, designed to lift and frame her bustline more dramatically under studio lights. While Russell later claimed she never wore the thing, Hughes’s reputation as a hyper-controlling, detail-obsessed innovator was sealed. He didn’t just direct movies—he reimagined how to shoot them.

But filmmaking was just the opening act. Hughes’s true passion—perhaps his purest love—was aviation. In 1935, he set a world airspeed record flying the Hughes H-1 Racer. In 1938, he flew around the globe in 91 hours, earning him a ticker-tape parade in New York and a congratulatory telegram from President Franklin D. Roosevelt. His company, Hughes Aircraft, exploded into a major defense contractor, developing radar systems, missiles, and later, aerospace technology. He personally test-piloted many of the prototypes—sometimes successfully, sometimes not.

The worst crash came in 1946 while piloting the XF-11 reconnaissance plane over Beverly Hills. He clipped telephone wires and crash-landed in a residential area, destroying several homes. He broke dozens of bones, suffered third-degree burns, and nearly died. He was pulled from the wreckage by a U.S. Marine who happened to live nearby. The physical pain lingered for the rest of his life. So did the emotional trauma.

This is the crash that many believe began driving Howard Hughes crazy.

He emerged from the hospital addicted to morphine, codeine, and later Valium. But the painkillers didn’t just numb the physical agony—they dulled the sharp edges of a mind that was becoming unhinged. He began displaying symptoms that today would be clearly diagnosed: Obsessive-Compulsive Disorder (OCD), Post-Traumatic Stress Disorder (PTSD) from repeated crashes, Traumatic Brain Injury (TBI) from head trauma, and likely undiagnosed neurosyphilis, which can cause hallucinations and severe personality changes in its late stages.

He began spiraling. He became consumed with hand-washing rituals that lasted hours. He insisted on sealed containers for his food. He wrote memos detailing the precise number of tissues someone should use when handling a document. He refused to be touched. And then, gradually, he refused to be seen at all.

By the 1950s, Hughes disappeared from public life. He moved into the Desert Inn hotel in Las Vegas and refused to leave. When the owners threatened eviction, he bought the hotel. Then he bought more—four additional Vegas properties, including the Sands and the Frontier. He watched the city from behind blackout curtains while seated naked in a chair, surrounded by jars of his own urine. He ate the same meal—TV dinners, Hershey bars, and whole milk—every day. For months at a time, he wouldn’t speak. He communicated through written notes. Many were borderline incoherent.

He trusted only a small inner circle of Mormon aides—dubbed the “Mormon Mafia.” These men controlled access to Hughes. They decided who could speak to him, when medications were administered, and even, allegedly, which documents he signed. Whether they were loyal caretakers or self-serving gatekeepers is still up for debate. Some say they protected him. Others believe they manipulated him for their own ends.

Meanwhile, Hughes was still making moves. His influence extended far beyond real estate and film. His company, Hughes Aircraft, was a key contractor for the U.S. government. In 1974, it was revealed that the CIA used Hughes’s name and company to build a deep-sea vessel—the Glomar Explorer—to recover a sunken Soviet submarine. The operation, known as Project Azorian, remains one of the most ambitious and secretive intelligence operations in history. Hughes’s name gave the cover story credibility. It also gave the CIA plausible deniability.

Hughes’s political entanglements didn’t stop there. He had longstanding financial connections to powerful people—most notably Richard Nixon. It’s widely believed that Hughes funneled large sums of money through intermediaries like Bebe Rebozo, a close Nixon ally. Some even argue that the 1972 Watergate break-in was partly motivated by a desire to retrieve sensitive documents linking Nixon to Hughes. Though never definitively proven, the rumors persisted and added another shadow to Hughes’s legacy.

And through it all, he was deteriorating—mentally, physically, and emotionally.

His fingernails grew inches long and curled under themselves. His toenails cracked and yellowed. He refused to bathe or cut his hair. He developed allodynia, a condition where even a soft touch causes extreme pain. He wore Kleenex boxes on his feet and sat naked for days at a time in darkened rooms, watching old movies on repeat. He feared germs, radiation, and even sunlight. His world shrank to a few rooms and a few carefully controlled interactions. He had gone from a bold aviator and innovator to a whisper behind a hotel room door.

In 1972, author Clifford Irving sold a fake Hughes autobiography to publisher McGraw-Hill. Irving claimed he had conducted secret interviews with Hughes. The hoax unraveled spectacularly when Hughes—out of hiding—called in to a press conference and publicly denied any involvement. The voice was unmistakably his. It was the last time the world would ever hear it.

In his final years, Hughes drifted from hotel to hotel, city to city: Managua, Vancouver, Acapulco, London. He traveled by private jet, hidden away, often sedated. His last known photograph is debated. Even his closest aides gave conflicting accounts of where he was at any given time.

On April 5, 1976, Howard Hughes died aboard a chartered Learjet, 30,000 feet over New Mexico, en route from Acapulco to Houston’s Methodist Hospital. He was pronounced dead at 1:27 a.m. The official cause: kidney failure. But when his body was examined, doctors were shocked. He weighed just 90 pounds and had shrunk more than four inches in height. His hair and beard were matted and uncut. His fingernails were several inches long. His skin was covered in sores. He was so unrecognizable, the FBI had to use fingerprints to identify him.

The coroner declared natural causes. But an 18-month private investigation painted a more disturbing picture. According to their report: “Persons unknown intentionally administered a deadly injection of codeine painkiller to this comatose man—obviously needlessly and almost certainly fatal.”

Was it euthanasia? Murder? A mercy killing? Or just gross negligence? We’ll likely never know. But Hughes’s legacy was immediately thrown into chaos. There was no clear will. Dozens of people claimed to have one. Most were forged. One, presented by gas station attendant Melvin Dummar, claimed Hughes had left him $156 million. It was ruled a fake, but the story became the basis for the film Melvin and Howard.

Even in death, Hughes was a myth waiting to be rewritten.

His Howard Hughes Medical Institute—originally established as a tax shelter—became one of the largest and most respected biomedical research organizations in the world. His story inspired books, films (The Aviator among them), and countless conspiracy theories. He remains one of the most complex, contradictory figures in American history.

So, what drove Howard Hughes crazy?

It wasn’t just the painkillers. Or the isolation. Or the crashes. It was the collision of genius without limits, power without oversight, and a mind without rest. He was a man of staggering vision—who could imagine worlds that hadn’t yet been built—but also a man whose compulsions devoured him from the inside out. He chased perfection in everything: flight, film, business, beauty. And perfection, for Hughes, was always just one more note, one more tweak, one more cleaning away.

He died not just from kidney failure—but from the failure of a peripheral support system that let a brilliant man collapse into exponential madness behind closed doors.

This is the real Howard Hughes—the boy genius, the master builder, the spy asset, the germ-fearing recluse, the paranoid mogul, and the man whose life and death still stir questions we may never answer.

And this was the crazy life and death of Howard Hughes.

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POST-TRAUMATIC STRESS DISORDER IS A NASTY BITCH

PTSD1CLast month another police officer took his own life after a lengthy battle with Post-Traumatic Stress Disorder. I’ve handled lots of suicide cases over the years, but this one hit close to home –  I knew Corporal Ken Barker. We’d worked together prior to the events which brought on Ken’s PTSD.

Ken was one of the best-liked, most approachable Royal Canadian Mounted Police members I ever met. He certainly wasn’t the stereotype who’d you think would suffer a PTSD mental illness. Wait – there’s no such thing as a stereotype PTSD sufferer and, yes, PTSD is a mental illness.

PTSD2There’s a higher awareness of PTSD today than back in the 1990’s when I was posted with Ken. Personally, I’ve experienced events as a cop and a coroner which should have brought on PTSD in me, but didn’t. I was very aware of the disorder and knew to recognize the signs. Also, I wasn’t scared to talk about PTSD and I think that’s the best form of prevention and treatment.

Today, I watch with caution as my son’s career in the Canadian Army unfolds and the suicide deaths of soldiers pile up into a national crisis. There are more Canadian soldiers who died of PTSD related suicides than were killed in ten years of active combat in Afghanistan.

So who is this Post-Traumatic Stress Disorder bitch?

Clinically, PTSD is classified as a trauma and stress related disorder stipulated in the Diagnostic and Statistical Manual of Mental Disorders IV. 

It’s simply summarized as:

1. Exposure to a traumatic event.

This includes both physical harm, or the risk of serious injury or death to self or others, and a response to the event that involved intense fear, horror, or helplessness. The traumatic event should be of a type that would cause significant symptoms of distress in almost anyone, and that the event was outside the range of usual human experience.

2. Persistent re-experiencing.

PTSD3One or more of these must be present in the victim: flashback memories, recurring distressing dreams, subjective re-experiencing of the traumatic event(s), or intense negative psychological or physiological response to any reminder of the traumatic event(s).

A. Persistent avoidance and emotional numbing.

PTSD4This involves a sufficient level of avoidance of stimuli associated with the trauma, such as certain thoughts or feelings, or talking about the event(s) and avoidance of behaviours, places, or people that might lead to distressing memories as well as the disturbing memories, dreams, flashbacks, and intense psychological or physiological distress. It includes the inability to recall major parts of the trauma(s), or decreased involvement in significant life activities as well as a decreased capacity (down to complete inability) to feel certain feelings, and an expectation that one’s future will be somehow constrained in ways not normal to other people.

B. Persistent symptoms of increased arousal not present before.

These are all physiological response issues, such as difficulty falling or staying asleep, or problems with anger, concentration, or hyper-vigilance. Additional symptoms include irritability, angry outbursts, increased startle response, and concentration or sleep problems.

C. Duration of symptoms for more than 1 month.

If all other criteria are present but 30 days have not elapsed, the individual is diagnosed with acute stress disorder. Anything longer would be considered chronic.

D. Significant impairment.

The symptoms reported must lead to clinically significant distress or impairment of major domains of life activity, such as social relations, occupational activities, or other important areas of functioning.

PTSD5Although most people with PTSD will develop symptoms within three months of the traumatic event, some people don’t notice any symptoms until years after. A major increase in stress, or exposure to a reminder of the trauma, can trigger symptoms to appear months or years later.

Who’s susceptible to PTSD?

Generally, at highest risk are those who experience traumatic events more frequently and for longer exposure. Combat personnel (soldiers, sailors, and airmen) are at the forefront, followed by emergency responders like police, firefighters, and medical professionals.

PTSD6There are other risk groups. Survivors of violent acts like sexual assault and attempted murder commonly experience post-traumatic stress. This extends to accident victims and witnesses of violent incidents.

What’s the medical reason for PTSD?

Three areas of the brain which control and administer PTSD have been identified. They’re the prefrontal cortex, the amygdala, and the medial prefrontal cortex.

Traumatic events cause an over-reactive adrenaline response, which creates deep neurological patterns in the brain.

PTSD7These patterns can persist long after the event that triggered the fear, making an individual hyper-responsive to future fearful situations. During traumatic experiences, the high levels of stress hormones secreted suppressed hypothalamic activity that may be a major factor toward the development of PTSD.

These biochemical changes in the brain and body differ from other psychiatric disorders such as major depression and bi-polar. Individuals diagnosed with PTSD respond more strongly to a dexamethasone suppression test than individuals diagnosed with clinical depressions.

PTSD8In addition, most people with PTSD also show a low secretion of cortisol and high secretion of catecholamines in urine with a norepinephrine / cortisol ratio consequently higher than comparable non-diagnosed individuals. This contrasts to the normal fight-or-flight response, in which both catecholamine and cortisol levels are elevated after exposure to stress.

Getting clinical – brain catecholamine levels are high and corticotropin concentrations are high. Together, these create an abnormality in the hypothalamic-pituitary-adrenal (HPA) axis.

The HPA axis is responsible for coordinating the hormonal response to stress. Given the strong cortisol suppression to dexamethasone in PTSD, HPA axis abnormalities are predicated on strong negative feedback inhibition of cortisol, itself likely due to an increased sensitivity of glucocorticoid receptors.

Translating this reaction to human conditions gives a patho-physiological explanation for PTSD by a maladaptive learning pathway to fear response through a hyper-sensitive, hyper-reactive, and hyper-responsive HPA axis.

PTSD9Low cortisol levels may also predispose individuals to PTSD and studies indicate that people that suffer from PTSD have chronically low levels of serotonin, which contributes to the commonly associated behavioral symptoms such as anxiety, ruminations, irritability, aggression, suicidality, and impulsivity. Serotonin also contributes to the stabilization of glucocorticoid production.

Insufficient dopamine levels in patients with PTSD can contribute to anhedonia, apathy, impaired attention and moto-skill defects. Increased levels of dopamine leads to psychosis, agitation, and restlessness.

Why are flashbacks so common in PTSD sufferers?

In a traumatic experience, the mind processes and stores the memory differently than it stores regular experiences.

Sensory information about the trauma – smells, sights, sounds, tastes, and the feel of things – is given high priority in the mind and is remembered as something threatening.

PTSD10Once this happens, whenever the sufferer is faced with a touch, a taste, a smell, a feel, or a sight that reminds them of the trauma, the memory (and the feeling of threat) comes back up and vivid memories or flashbacks about the trauma occur.

Getting all clinical again, a hyper-responsiveness in norepinephrine receptors in the prefrontal cortex is connected to the flashbacks. A decrease in other norepinephrine functions prevents the memory mechanisms in the brain from processing that the experience and emotions the person is experiencing during a flashback are not associated with the current environment. In other words, it takes them right back to the trauma time and it seems very, very real.

What can be done about it?

Many sufferers feel guilt or shame around PTSD because they’re often told they should just ‘suck-it-up’ to get over difficult experiences. Others feel embarrassed in talking with others. Some feel like it’s somehow their own fault.

Here’s the common treatments.

Counselling

PTSD11Cognitive-behavioural therapy (CBT) is effective. Very effective. CBT teaches how thoughts, feelings, and behaviours work together and how to deal with problems and stress. Relaxation techniques, such as meditation and hypnosis are used. This exposure therapy helps the sufferer talk about their experience and helps reduce avoidance.

In my experience, this stuff works. But the sufferer has to know the disorder before accepting the treatment.

Medication

A number of medications can prevent PTSD or reducing its incidence, especially when given in close proximity to a traumatic event. These include:

SSRIs (Selective Serotonin Reuptake Inhibitors)

SSRIs are considered to be a first-line drug treatment. They include citalopram, escitalopram, fluoxetine, paroxetine, and sertraline.

Tricyclic antidepressants 

Amitriptyline benefits distress and avoidance symptoms. Imipramine is effective for intrusive symptoms.

Alpha-adrenergic antagonists

In a study of combat veterans, prazosin shows substantial benefit in relieving or reducing nightmares. Clonidine helps with startle, hyper-arousal, and general autonomic hyper-excitability.

Anti-convulsants, mood stabilizers, and anti-aggression agents

PTSD12Carbamazepine reduces arousal symptoms involving noxious affect, as well as mood or aggression factors. Topiramate is effective in achieving major reductions in flashbacks and nightmares. Zolpidem proves useful in treating sleep disturbances. Lamotrigine reduces re-experiencing symptoms as well as avoidance and emotional numbing. Valproic acid reduces symptoms of irritability, aggression, impulsiveness, and reducing flashbacks. Similarly, lithium carbide works well to control mood and aggressions (but not anxiety) symptoms. Buspirone has an effect similar to lithium, with the additional benefit of reducing hyper-arousal symptoms.

Antipsychotics

Risperidone is the main medication for dissociation, mood issues, and aggression issues while cyproheptadine, a serotonin antagonist, helps with sleep disorders and nightmares.

Atypical antidepressants

Nefazodone works with sleep disturbance symptoms, secondary depression, anxiety, and sexual dysfunction symptoms. Trazodone reduces or eliminates problems with anger, anxiety, and disturbed sleep.

Beta Blockers 

Propranolol has demonstrated possibilities in reducing hyper-arousal symptoms, including sleep disturbances – but the jury’s out.

Benzodiazepines

PTSD13These drugs are not recommended by clinical guidelines for the treatment of PTSD due to a lack of evidence of benefit. Nevertheless, some doctors use benzodiazepines with caution for short-term anxiety relief of hyper-arousal and sleep disturbance, and believe that the use of benzodiazepines is proper for acute stress, as this group of drugs promotes dissociation and ulterior revivals. While benzodiazepines can alleviate acute anxiety, there is no consistent evidence that they can stop the development of PTSD, or are at all effective in the treatment of posttraumatic stress disorder.

Additionally, benzodiazepines may reduce the effectiveness of psychotherapeutic interventions, and there’s some evidence that benzodiazepines contribute to the development and chronification of PTSD. Other drawbacks include the risk of developing a benzodiazepine dependence and withdrawal syndrome. Additionally, individuals with PTSD are at an increased risk of abusing benzodiazepines.

Glucocorticoids

High-dose corticosterone administration was recently found to reduce ‘PTSD-like’ behaviours in a rat models. In this study, corticosterone impaired memory performance, suggesting that it may reduce risk for PTSD by interfering with consolidation of traumatic memories. The neurodegenerative effects of the glucocorticoids, however, may prove this treatment counterproductive.

That’s great lab-rat stuff which I’m not going to try myself. However, the next stuff is something that I think ‘where’s there’s smoke – there’s fire”.

Cannabis

PTSD14There’s a study underway between a University and one of Canada’s largest producers of medicinal cannabis, suggesting that the active ingredients in marihuana – tetrahydrocannabinol and cannabinoids – may be very effective in reducing PTSD symptoms. Many PTSD sufferers self-medicate through black-market cannabis and swear by it. It’ll be interesting to see this clinical study’s results.

Support groups

PTSD15Support groups definitely help. Here people share experiences and learn from others. Connecting with people who understand what the sufferer goes through is probably the most effective form of treatment and this leads to identifying other forms of treatment such as medication and psychological intervention.

PTSD awareness is much greater in the twenty-first century, but the disorder is long known and buried. Historically they called it battle fatigue, shell-shock, and the thousand-yard stare. Soldiers were actually shot by their own command for perceived cowardness. I’ll bet the majority weren’t afraid – they were just suffering a nasty bitch of a disorder.

PTSD16On a personal note – looking back – I believe my dad suffered from PTSD. He was a gunner on a RCAF Lancaster bomber during World War II; the veteran of 113 operational runs. If that doesn’t do something to the psyche, I don’t know what would. I remember him sitting for long periods… on a big flat rock in our yard… in that thousand-yard stare… until his cigarette… burned down to his fingers… and snapped him back to reality.

After nearly six decades of life experience and being exposed to more traumatic life & death exposures than I can count, I can’t say that I’ve experienced PTSD.

Grief, yes. Compassion; in spades. Fear – I’ve been absolutely shit-scared, bewildered, and abhorred; being down on my belly under gunfire and questioning the existence and authority of Infinite Intelligence. But I’ve never experienced guilt and I don’t know much about it. Guilt seems like an evil, degenitive force who’s metaphysical purpose is to destruct. A lot more needs to be known about the psychological effects of guilt.

I think guilt is the nasty bitch in PTSD and I think that guilt walks hand-in-hand with shame.

Post-Traumatic Stress Disorder is a complex mix of psychological, physiological, and metaphysical workings and it’s nothing to be guilty about or ashamed of. It’s a naturally-occurring, mental illness. With proper support and effective treatment, PTSD sufferers can fully recover.

Remember, PTSD isn’t about what’s wrong. It’s about what’s happened.

Please leave your comments, ask questions, or tell about your experiences. It’s okay to talk about PTSD and raising awareness is the best form of treatment… and prevention.